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Quercetin Prevents Antioxidant Damage by Eliminating ROS It is known that quercetin can scavenge ROS, and most of the oxidative damage in vivo is attributed to ROS, so quercetin can resist oxidative damage, such as radiation-induced ultraviolet radiation B (UVB) skin lesions, respiratory damage, and other oxidative damage diseases [25]

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It was demonstrated that ferroptosis is highly correlated with cardiomyocyte death, and mammalian target of rapamycin (mTOR) plays an important role in protecting cardiomyocytes against excess iron and ferroptosis by inhibiting the production of lipid-derived ROS [105]