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These molecules are observed to have rising concentrations during pregnancy (117) ( Figure 2 )
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Supporting this, Yang and his team demonstrated that EGCG, at concentrations between 5 and 25 µmol/L, could prevent the activation of p38 MAPK and JNK1/2 in HUVECs exposed to angiotensin II, a compound known to induce endothelial dysfunction
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