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In another study, it was shown that the decreases in GABAergic neuronal density seen in the pathogenesis of schizophrenia, abnormalities in receptors, and reuptake areas were corrected with GABA agonists, and thus the cognitive symptoms of schizophrenia patients could be improved

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To overcome this, a study developed monocyte membrane-coated nanoparticles (MoNP) for targeted delivery to activated, inflamed endothelium on plaque surfaces, effectively reducing plaque development in mice without causing significant histopathological changes in major organs [194]